Base de dados : HANSEN Pesquisa : NERVOS PERIFERICOS/PATOL [Descritor de assunto] Referências encontradas : 37 [refinar] Mostrando: 1 .. 20   no formato [Detalhado]

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Id: 27312 Autor: McDougall, A. Colin. Título: Bacillary and histopathological findings in the peripheral nerves of armadillos experimentally infected with M. leprae. Fonte: Int. J. Lepr;65(2):260-261, Jun. 1997. . Descritores: Mycobacterium leprae/citol Mycobacterium leprae/patogen Nervos Periféricos/irrig Nervos Periféricos/microbiol Nervos Periféricos/patol Limites: Animais Localização: BR191.1

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Id: 27308 Autor: Pandya, Shubhada S. Título: An anatomist in leprosyland. Fonte: Int. J. Lepr;65(2):246-251, Jun. 1997. ilus, tab. Descritores: Hanseníase/hist Hanseníase/patol Nervos Periféricos/patol Localização: BR191.1

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Id: 27149 Autor: Tzue-Chun, Liu; Zhong-Min, Ji; Skinsnes, Olaf K. Título: Light and electron-microscopic study of M. leprae infected Armadillo Neves. Fonte: Int J Lep;57(1):65-72, Mar. 1989. ^bilus, ^btab. Resumo: Lesions in peripheral nerves of armadillos experimentally infected with Mycobacterium leprae were studied by light- and electron-microscopy. Bacilli could be found clearly inside axons of unmyelinated nerve fibers. Heavily bacillated Schwann cells were seen embracing unmyelinated axons with interrupted cytoplasmic membranes. This indicated the initiation of rupture of those cells which were responsible for the liberation of bacilli into the axons. The nerve lesions were divided into three grades according to their severity: grade I showed lesions focalized in the perineurium; grade II lesions were scattered inside nerve tissue; and in grade III lesions the nerve tissues were diffusely affected. No regressive changes, such as fibrosis or scar formation, were seen in the nerve lesions. Bacillated macrophages were not as foamy as those of human lesions, indicating that these bacillated cells were younger or more easily disrupted with a higher turnover than the cells in human lesions. This would promote the spread of lesions in armadillos, and would explain the less foamy appearance of the cells. We found bacilli inside lymphatics surrounding the nerves, substantiating the opinion that lesions spread to peripheral nerves not only by a hematogenous route but also by the lymphatics^ien. Descritores: Tatus/microbiol Nervos Periféricos/microbiol Nervos Periféricos/patol Nervos Periféricos/ultraest Mycobacterium leprae/imunol Limites: Animais Meio Eletrônico: http://hansen.bvs.ilsl.br/textoc/revistas/intjlepr/1989/pdf/v57n1/v57n1a09.pdf - en. Localização: Br191.1

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Id: 25468 Autor: Carayon, A Título: Bilan de recherches physiopathologiques sur la nevrite lepreuse: 1-role de la temperature, des microtraumatismes par elongation ou subluxation nerveuse et de la striction canalaire ?- Fonte: s.l; s.n; 1977. 18p p. ilus. Resumo: (Role of temperature, microtraumatisms by elongation or subluxation and canalar stricture.) Cold has a slight aggravating effect on leprosy which is observed in some countries as Iran, South Russia, North India and the Andean cordillera. The subluxation of the ulnar nerve has facilitating effect in the development of the neuritic damage in a limited number of patients. The elongation is important for the production of the ulnar neuritis. The canalar stricture is a major factor causing nerve damage in leprosy. It explains the segmentary localization of the principal damages of the leprous nerves in the proximal areas situated above the tunnels. Intraneural hypertension appears first and is responsible for the swelling of the nerve which gets entraped inside the tunnel. A circulus viciosus is, then, created which maintains and aggravates the intraneural hypertension. Descritores: HANSENIASE DIMORFA/compl HANSENIASE DIMORFA/imunol HANSENIASE DIMORFA/microbiol HANSENIASE DIMORFA/patol HANSENIASE DIMORFA/fisiopatol HANSENIASE VIRCHOWIANA/compl HANSENIASE VIRCHOWIANA/imunol HANSENIASE VIRCHOWIANA/microbiol HANSENIASE VIRCHOWIANA/patol HANSENIASE VIRCHOWIANA/fisiopatol HANSENIASE TUBERCULOIDE/compl HANSENIASE TUBERCULOIDE/imunol HANSENIASE TUBERCULOIDE/microbiol HANSENIASE TUBERCULOIDE/patol HANSENIASE TUBERCULOIDE/fisiopatol NEURITE/etiol - SINDROMES DE COMPRESSAO NERVOSA/etiol  NERVOS PERIFERICOS/les  NERVOS PERIFERICOS/patol  NERVOS PERIFERICOS/fisiopatol Limites: HUMANO Localização: BR191.1; 00508/s

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Id: 24738 Autor: Carayon, A; Giraudeau, P Título: La neurolyse fasciculaire dans la lepre 20 ans apres (1957-1977) ?- Fonte: s.l; s.n; 1977. 25p p. ilus. Resumo: A better knowledge of the pathological and physiopathological nervous involvement in leprosy and a more extended experience of fascicular neurolysis have proved the value of this procedure proposed by Carayon in 1957. The opposed opinions are discussed and the authors give the strict rules required for a good indication and a good surgical technic according to the kind of damage and to the nerve concerned. Descritores: NERVOS PERIFERICOS/les NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/cirurg HANSENIASE/compl HANSENIASE/cirurg - NEUROCIRURGIA/métodos  NEUROCIRURGIA/reabil Limites: HUMANO Localização: BR191.1; 00533/s

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Id: 24518 Autor: Chimelli, Leila. Título: Valor da biópsia de nervo no diagnóstico da hanseníase - auxílio à clínica nos casos de forma neural pura e reativação / Value of the biopsy of nerve in the diagnosis of leprosy - aid to the clinic in the cases of pure neural form and reactivation Fonte: Hansen. int;(n.esp):89-92, Jun. 1998. . Descritores: HANSENIASE/patol HANSENIASE/cirurg HANSENIASE/diag NERVOS PERIFERICOS/anorm NERVOS PERIFERICOS/anat NERVOS PERIFERICOS/citol NERVOS PERIFERICOS/les NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/cirurg Limites: RELATO DE CASO ESTUDO COMPARATIVO HUMANO Localização: BR191.1

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Id: 23141 Autor: Garbino, Jose Antonio; Ura, Somei; Belone, Andrea de Faria Fernandes; Marciano, Lucia Helena Soares Marciano; Fleury, Raul Negrao. Título: Clinical and diagnosis aspects of the primarily neural leprosy. Fonte: Hansen. int;29(2):130-136, Jul.-Dez. 2004. ilus, tab, graf. Resumo: A total of 33 patients, 28 males and five females, from nine to 87 years of age, with suspected leprosy associated peripheral neuropathy, without detectable skin lesion or positive skin bacilloscopy, were studied during the period of 1994 to 2004. Patients were submitted to dermatological and neurological examination, electrophysiologic tests, Mitsuda reaction and nerve biopsy. Samples for histopathological exams were stained with hematoxillin-eosin, Faraco-Fite and immunohistochemistry with anti- BCG antibodies. Among patients with suspected leprosy, the clinical presentation of polyneuropathy occurred in 17 (51,51%) patients while 13 (39,39%) presented mononeuropathy multiplex and 3 (9,1%) mononeuropathy. The hematoxillin-eosin and Faraco-Fite stainings confirmed the leprosy diagnosis in 10 (30,30%) patients. Three patients presented a borderline pattern, two tuberculoid and no characteristic histological pattern was observed in the remaining five patients. The final calssification depended on the clinical-histological correlation. Immunohistochemistry increased the diagnosis to 11 (33,33%) cases. Among the remaining 22 patients, three patients had leprosy confirmed, one was clinically understood as a primarily neural leprosy, probably tuberculoid form of the childhood, increasing the diagnosis to 14 (42,43%) cases. From the remaining patients, 19 (57,57%) were excluded during the follow-up. The primarily neural leprosy (PNL) is an unusual leprosy presentation and a complex form to diagnose. The clinical follow-up accompanied by the improvement of histopathological examination of the nerve may add more accuracy to the investigation of the suspected leprosy neuropathies (AU). Descritores: HANSENIASE/compl HANSENIASE/diag NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/fisiopatol Meio Eletrônico: http://hansen.bvs.ilsl.br/textoc/hansenint/v21aov29/2004/PDF/v29n2/v29n2a05eng.pdf - en. Localização: BR191.1

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Id: 23140 Autor: Garbino, Jose Antonio; Ura, Somei; Belone, Andrea de Faria Fernandes; Marciano, Lucia Helena Soares Marciano; Fleury, Raul Negrao. Título: Aspectos clinicos e diagnosticos da hanseniase primariamente neural / Clinical and diagnostic aspects of the primarily neural leprosy Fonte: Hansen. int;29(2):124-129, Jul.-Dez. 2004. ilus, tab, graf. Resumo: Neste trabalho foram acompanhados clinicamente 33 pacientes com a idade de nove a 87 anos, 28 masculinos e cinco femininos, suspeitos de hanseníase, com neuropatia periférica, sem lesão de pele evidente e sem baciloscopia e biópsia de pele positivas, durante o período de 1994-2004. Todos foram submetidos aos exames dermatológico, neurológico, neurofisiológico, reação de Mitsuda e biópsia de nervo. As biópsias de nervo foram fixadas em FMA e ósmio e os cortes histológicos corados pela hematoxilina-eosina, Fite-Faraco e imunoistoquímica com anticorpo anti-BCG. O quadro clínico de polineuropatia ocorreu em 17 (51,51%) pacientes, mononeuropatia múltipla em 13 (39,39%) e mononeuropatia em 3 (9,1%). As colorações pela hematoxilina-eosina e o Fite-Faraco foram conclusivas para hanseníase em 10 (30,30%) pacientes, três deles apresentaram padrão Dimorfo, dois padrão Tuberculóide e nos restantes cinco não havia padrão histológico característico. A classificação final dependeu da correlação clínico-histológica. O estudo imunoistoquímico foi positivo em todos os casos com infiltrado inflamatório específico, e em um paciente com infiltrado inflamatório inespecífico, aumentando o diagnóstico de hanseníase para 33,33% (11). Durante o acompanhamento clínico dos 22 pacientes restantes, três tiveram o diagnóstico de hanseníase confirmado e nos demais 19 (57,57%) foi afastada a possibilidade de hanseníase, passando a 42,43% (14) os pacientes com hanseníase primariamente neural. A hanseníase primariamente neural é uma manifestação incomum da hanseníase, e o seu diagnóstico é complexo. O acompanhamento clínico em conjunto com a melhora das técnicas do exame histopatológico do nervo oferece mais acuidade para a investigação dos casos suspeitos de hanseníase primariamente neural (AU). Descritores: HANSENIASE/compl HANSENIASE/diag NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/fisiopatol Meio Eletrônico: http://hansen.bvs.ilsl.br/textoc/hansenint/v21aov29/2004/PDF/v29n2/v29n2a05.pdf - pt. Localização: BR191.1

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Id: 22874 Autor: Anon Título: Thérapeutique chirurgicale dans da lèpre ..- Fonte: s.l; s.n; s.d. 59 p. ilus. Descritores: HANSENIASE/compl HANSENIASE/cirurg NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/cirurg Limites: RELATO DE CASO Localização: BR191.1; 00534/s

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Id: 22632 Autor: Antunes, Sergio Luis Gomes Título: Estudo imuno-histoquímico e ultra-estrutural da inervação de lesões cutâneas de hanseníase em fase inicial ?- Fonte: Niterói; s.n; 1994. 159 p. ilus, tab, graf. Tese: Apresentada a Universidade Federal Fluminense para obtenção do grau de Doutor. Resumo: A inervação cutanea das lesões da hanseníase em fase inicial foi estudada através da expressão imuno-histoquímica das proteinas neuronais NGER ("nerve growth factor receptor"). PGP 9.5 ("protein gene product 9.5") ENE (enolase neurônio-específica") e também através do estudo ultra-estrutural com microscopia eletronica de transmissão. Observamos uma redução do numero de fibras nervosas PGP 9.5- e ENE-positivas não estava relacionada topograficamente com o infiltrado inflamatório. No estudo ultra-estrutural, a maioria dos nervos examinados mostrava-se sem envolvimento inflamatório e sem alterações morfológicas. O infiltrado inflamatório estava presente em sete nervos e em seis deles, situava-se em torno do perincuro. Em apenas um nervo permeava as lamelas perineurais e invadia o endoneuro. Mesmo alcançando o endoneuro, as células inflamatórias não mostravam interação de contacto com a fibra nervosa. Não encontramos alterações morfológicas das fibras ocasionadas pela inflamação. A existência de redução do número de fibras nervosas e de fibrose endoneural independentes do evento inflamatorio foi suspeitada. Existe portanto, nas fases iniciais da hanseniase, uma alteração seletiva na expressão de proteinas neuronais não relacionada com o incipiente infiltrado específico. A nivel ultra-estrutural, não detectamos lesões das fibras nervosas diretamente causadas pelo infiltrado inflamatório, este comprometia mais o componente mesenquimatoso do nervo (perineuro). O infiltrado hanseniano inicial não deve ser implicado como o unico responsavel pelos distúrbios neurológicos exibidos pelos pacientes hansenianos. A existência de um processo patogênico distinto da inflamação e intrinseco à fibra nervosa, foi cogitada para explicar os mecanismos de lesão neural da neuropatia hanseniana. (AU). Descritores: HANSENIASE/patol PATOLOGIA NERVOS PERIFERICOS/imunol NERVOS PERIFERICOS/patol Limites: RELATO DE CASO Localização: BR191.1; WC335.103, A89e

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Id: 20625 Autor: Ermakóva, Nína. Título: Studies on leprosy. II. Formation, distribution and diagnostic significance of pigment. Fonte: Int. J. Lepr;4(4):445-454, Oct.-Dec. 1936. ilus. Resumo: An iron-containing blood pigment was found in the affected organs and tissues of all cases of leprosy that I examined. In the nodular form of the disease this pigment is deposited in strikingly large quantities, while in the maculo-neural and tuberculoid forms the quantity is very small. The cause of the pigment formation may be (a)erythrocyte destruction in the general blood stream, or (b) some process of local character. The generally accepted view that the leprosy bacillus does not excrete toxic substances that may act on the cells of the organisms seems to be against the first of these theories. However, this view is based entirely on clinical data and not on experimental immuno-biological studies. It seems quite possible that products of bacillary activity may be fixed by the lipoid membrane of the erythrocyte and destroy them. In favor of the second theory, that the pigment is of local origin, is my observation that erythrophagocytosis is found with some constancy in the lesions. It is true that in cases of the nodular form phagocytosis is demonstrated with great difficulty, though the pigment is deposited in large quantities. However, in some of the cases in which there were lesser quantities of pigment, that did not interfere with the clearness of the picture, phagocytosis could be observed. At any rate, the question must be regarded as an open one, requiring further study... (AU). Descritores: HANSENIASE/clas HANSENIASE/diag HANSENIASE/patol HANSENIASE VIRCHOWIANA/sangue HANSENIASE VIRCHOWIANA/patol HANSENIASE TUBERCULOIDE/sangue HANSENIASE TUBERCULOIDE/patol HANSENIASE/sangue HANSENIASE/compl - PELE  NERVOS PERIFERICOS/patol  BACO/patol  FIGADO/patol  GÔNADAS/patol  LINFONODOS/patol  HEMOSSIDERINA Limites: HUMANO necropsia biopsia Localização: BR191.1

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Id: 18492 Autor: Turkof, Edvin; Richard, Bruce; Assadian, Ojan; Khatri, Bharat; Knolle, Erich; Lucas, Sebastian Título: Leprosy affects facial nerves in a scattered distribution from the main trunk to all peripheral branches and neurolysis improves muscle function of the face ..- Fonte: s.l; s.n; 2003. 8 p. ilus, tab, graf. Resumo: Current literature rejects nerve release in leprous facial neuropathy and states that lesions are restricted to the peripheral zygomatic branches. Since there are approximately 500,000 patients with this disease throughout the world, we wanted to clarify the precise location of facial nerve's affection and the benefit of neurolysis. Our study showed that in patients with leprosy, the facial nerve's main trunk, the peripheral zygomatic branches, and all other branches were affected. Follow-up showed improvement in lagophthalmos and in misreinnervation, with no improvement in the control cohort. Nerve release improves muscle function in leprous facial neuropathy, provided surgery is performed on all affected segments. Intraoperative electroneurodiagnostics is an effective tool for detecting the most proximal site of lesion and ensuring effective surgery. (AU). Descritores: ESTUDOS DE COORTES ESTIMULACAO ELETRICA ELETROFISIOLOGIA MUSCULOS FACIAIS/inerv MUSCULOS FACIAIS/fisiol NERVO FACIAL/patol NERVO FACIAL/fisiol NERVO FACIAL/fisiopatol HANSENIASE DIMORFA/patol HANSENIASE DIMORFA/fisiopatol HANSENIASE DIMORFA/cirurg NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/cirurg RESULTADO DE TRATAMENTO - PERIODO INTRA-OPERATORIO  CONDUCAO NERVOSA Limites: HUMANO MASCULINO FEMININO ADULTO MEIA-IDADE SUPPORT, NON-U.S. GOV'T Meio Eletrônico: - . Localização: BR191.1; 09072/s

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Id: 18130 Autor: Boddingius, J Título: Ultrastructural changes in blood vessels of peripheral nerves in leprosy neuropathy ..- Fonte: s.l; s.n; September 26, 1977. 19 p. ilus, tab. Resumo: The ultrastructure of blood vessels in endoperi and epineurium was studied in peripheral cutaneous nerve biopsies of 16 borderline (BB), borderline-lepromatous (BL) or lepromatous (LL) leprosy patients some of whom were in reversal reaction. Comparable vessels in nerve biopsies of control cases and vessels in skin lesion biopsies of the leprosy patients were also studied. Vascular changes were found in nerves of all the leprosy patients. The changes were pronounced in endoneurial vessels and affected 1. endothelial continuity and surface structure, 2. basement membranes od endothelium and pericytes, and 3. the vessel lumen. In addition, intra-endothelial (IE) Mycobacterium leprae were a feature in some of the patients. Gaps occurring between endothelial cells and plasma insudation both noticed in vessels of fascicles with early to very early neuropathy suggested extensive leakage which, in all propability, causes early nerve fibre damage. Luminal and abluminal endothelial protusions, which were frequently observed, may enhance transendothelial transposrt. Fenestrations and endothelial attenuation, possibly, lead to an increase in vascular permeability. Endothelial phagocytotic activity, particularly in small (epineural) arteries, appeared to be stimulated, possibly, by circulating M. Leprae. Basement membrane multilayering (a hyaline zone) was found peripherally to pericytes, as is the case in tuberculoid leprosy (Boddingus, 1976). In a number of patients, multilayering occurred also periendothelially. Perivascular zones, which are thought to initiate or aggravate neuropathological changes by impairment of diffusion of oxygen and nutrients or metabolites, were very wide in endoneurial vessels of patients in reversal reaction and this suggested an immunological aetiology. Partial or total vessel lumen occlusion, seen in advanced lepromatous neuropathy, most likely contributes to final nerve fibre degeneration and endoneurial fibrosis. M. leprae were found intra-endothelially in endoneurial vessels, though only fascicles with advanced neuropathy whereas bacilli were not seen in vessel lumina. By contrast, infascicles with realtively early neuropathy, solid (viable) bacilli were frequently encountered intra-axonally in myelinated fibres. This suggested thast, inmany instances, primary infiltration of M. leprae into nerve facicles may arise from intraaxonal..(AU). Descritores: PERMEABILIDADE CAPILAR HANSENIASE/patol DEGENERACAO NEURAL NERVOS PERIFERICOS/patol DOENCAS DO SISTEMA NERVOSO PERIFERICO/patol FAGOCITOSE VASA NERVORUM/patol PELE/inerv Limites: HUMANO MASCULINO FEMININO CRIANÇA ADULTO MEIA-IDADE ADOLESCENTE Meio Eletrônico: - . Localização: BR191.1; 00031/s

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Id: 18043 Autor: Shetty, V. P; Mehta, L. N; Antia, N. H; Irani, P. F Título: Teased fibre study of early nerve lesions in leprosy and in contacts, with electrophysiological correlates ..- Fonte: s.l; s.n; jul. 1977. 4 p. ilus, tab. Resumo: A teased fibre technique was used to study 19 biopsies of the index finger branch of the radial cutaneous nerve of leprosy patients and contacts. These were compared with four normal nerves. Five nerves were from patients with preclinical nerve lesions, five from leprosy patients with minimal sensory nerve impairment, and five from contacts of lepromatous leprosy. The extent of demyelination in preclinical nerve lesions in leprosy and in contacts of leprosy is recorded. The usefulness of nerve conduction velocity studies in early leprosy patients and in contacts is discussed.(AU). Descritores: HANSENIASE/patol HANSENIASE/fisiopatol FIBRAS NERVOSAS MIELINIZADAS CONDUCAO NERVOSA DEGENERACAO WALLERIANA NERVOS PERIFERICOS/patol NERVOS PERIFERICOS/fisiopatol Limites: HUMANO MASCULINO FEMININO ADULTO ADOLESCENTE Meio Eletrônico: - . Localização: BR191.1; 01881/s

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Id: 17809 Autor: Antia, Noshir H Título: The significance of nerve involvement in leprosy ..- Fonte: s.l; s.n; jul. 1974. 9 p. ilus. Resumo: The significance of nerve involvement in leprosy is discussed. The treatment is outlined and the surgical technique is described.(AU). Descritores: HANSENIASE/compl HANSENIASE/quimioter HANSENIASE/patol HANSENIASE/cirurg PARALISIA/etiol NEURITE/quimioter NEURITE/etiol NEURITE/patol NEURITE/cirurg NERVOS PERIFERICOS/patol Limites: HUMANO Meio Eletrônico: - . Localização: BR191.1; 02020/s

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Id: 17808 Autor: Job, C. K Título: Mechanism of nerve destruction in tuberculoid-borderline leprosy ..- Fonte: s.l; s.n; sep. 1973. 14 p. ilus. Resumo: The ultrastructural changes in radial cutaneous nerve biopsies from 3 tuberculoid and 14 tuberculoid-borderline patients were studied. In tuberculoid leprosy the nerve parenchyma was almost entirely replaced by tuberculoid granuloma. In nerves from borderline-tuberculoid leprosy many axons showed segmental demyelination and Wallerian degeneration. The nerve tissue was gradually replaced by proliferating collagen fibrils and inflammatory granulomata. M. leprae were rare and when present they were found in phagolysosomes of Schwann cells and macrophages. Schwann cells were surrounded and occasionally infiltrated by lymphocytes and macrophages. It was suggested that "sensitized Schwann cells" were the target of a hypersensivity reaction resulting in their degeneration or death which in turn produced segmental demyelination or Wallerian degeneration.(AU). Descritores: HANSENIASE/imunol HANSENIASE/microbiol HANSENIASE/patol DOENCAS DESMIELINIZANTES ANTIGENOS DE BACTERIAS MICROSCOPIA ELETRÔNICA MYCOBACTERIUM LEPRAE/isol NERVOS PERIFERICOS/patol CELULAS DE SCHWANN Limites: HUMANO Meio Eletrônico: - . Localização: BR191.1; 02021/s

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Id: 17791 Autor: Dayan, A. D; Sandbank, U Título: Pathology of ther peripheral nerves in leprosy: report of a case ..- Fonte: s.l; s.n; oct. 1970. 6 p. ilus, graf. Resumo: Peripheral nerve trunks were examined from a case of lepromatous leprosy. M. leprae was seen in the nerves in several types of cells. isolated nerve fibre preparations showed demyelinating lesions due to Schwann cell damage and complete nerve fibre degeneration. The causes of this mixed pattern of damage may involve both a direct effect of the bacilli on nerve fibres and various epiphenomena.(AU). Descritores: DOENCAS DESMIELINIZANTES/patol HANSENIASE/patol NERVO FEMORAL/patol AUTOPSIA NERVOS PERIFERICOS/patol CELULAS DE SCHWANN NERVO CIATICO/patol DEGENERACAO NEURAL NERVO ULNAR/patol REGENERACAO NERVOSA Limites: HUMANO IDOSO MASCULINO Meio Eletrônico: - . Localização: BR191.1; 02018/s

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Id: 16380 Autor: Habib, Ali Aamer; Mozaffar, Tahseen Título: The neuropathology of leprosy ..- Fonte: s.l; s.n; Jan., 2002. 3 p. ilus. Descritores: HISTORIA DA MEDICINA DO SÉCULO 19 HISTORIA DA MEDICINA DO SÉCULO 20 HANSENIASE HANSENIASE NERVOS PERIFÉRICOS RETRATOS Localização: BR191.1; 08654/s

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Id: 15793 Autor: Shetty, V. P; Uplekar, M. W; Antia, N. H Título: Immunohistological localization of mycobacterial antigens within the peripheral nerves of treated leprosy patients and their significance to nerve damage in leprosy ..- Fonte: s.l; s.n; 1994. 7 p. ilus, tab. Descritores: HANSENIASE HANSENIASE HANSENIASE ANTICORPOS MONOCLONAIS ANTIGENOS DE BACTÉRIAS TÉCNICAS IMUNOENZIMATICAS MYCOBACTERIUM LEPRAE MYCOBACTERIUM LEPRAE NERVOS PERIFÉRICOS NERVOS PERIFÉRICOS NERVOS PERIFÉRICOS Localização: BR191.1; 06970/s

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Id: 15757 Autor: Habib, Ali Aamer; Mozaffar, Tahseen Título: The neuropathology of leprosy ..- Fonte: s.l; s.n; 2002. 3 p. ilus. Descritores: HANSENIASE HANSENIASE HISTORIA DA MEDICINA DO SÉCULO 19 HISTORIA DA MEDICINA DO SÉCULO 20 NERVOS PERIFÉRICOS RETRATOS Localização: BR191.1; 08447/s

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3	and or and not		PalavrasPalavras do títuloPalavras do resumoAutorAutor institucionalDescritor de assuntoLimitesIdiomaPaís, ano de publicaçãoRevistaTipo de literaturaNível bibliográficoMonografia em sérieTipo de publicaçãoConferênciaIdentificador únicoLocalizaçãoSuporte eletrônicoData de entradaTipo de registro

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